Cerebral Infarction

Cerebral Infarction yog dab tsi?

Cerebral infarction tseem hu ua ischemic stroke, nws yog kev puas tsuaj ntawm cov ntaub so ntswg ntawm lub hlwb tom qab cerebral artery occlusion, uas tej zaum yuav nrog los ntshav.Lub pathogenesis yog thrombosis lossis embolism, thiab cov tsos mob sib txawv nrog cov hlab ntsha koom nrog.Cerebral infarction suav txog 70% - 80% ntawm tag nrho cov mob stroke.

Dab tsi yog Etiology ntawm Cerebral Infarction?

Cerebral infarction yog tshwm sim los ntawm kev txo qis lossis nres ntawm cov ntshav ntws hauv cov ntshav hauv zos cov hlab ntsha ntawm cov ntaub so ntswg ntawm lub paj hlwb, ua rau cov ntaub so ntswg ischemia thiab hypoxia hauv cov ntshav, ua rau lub hlwb necrosis thiab softening, nrog rau cov tsos mob thiab cov tsos mob. ntawm cov khoom sib xws, xws li hemiplegia, aphasia, thiab lwm yam kab mob neurological deficit.

Yam tseem ceeb

Ntshav siab, kab mob plawv, ntshav qab zib, rog rog, hyperlipidemia, noj rog, thiab tsev neeg keeb kwm.Nws muaj ntau dua nyob rau hauv cov neeg laus thiab cov laus hnub nyoog 45-70.

Cov tsos mob ntawm Cerebral Infarction yog dab tsi?

Cov tsos mob ntawm cerebral infarction yog complex, nws muaj feem xyuam rau qhov chaw ntawm lub hlwb puas, qhov luaj li cas ntawm cerebral ischemic hlab ntsha, qhov hnyav ntawm ischemia, seb puas muaj lwm yam kab mob ua ntej pib, thiab seb puas muaj kab mob muaj feem xyuam rau lwm yam tseem ceeb hauv nruab nrog cev. .Hauv qee qhov mob me, tej zaum yuav tsis muaj cov tsos mob tshwm sim, uas yog, asymptomatic cerebral infarction Tau kawg, kuj tseem tuaj yeem tshwm sim limb tuag tes tuag taw lossis vertigo, uas yog, kev tawm tsam ischemic.Hauv qee qhov mob hnyav, yuav tsis yog limb tuag tes tuag taw xwb, tab sis txawm mob coma lossis tuag.

Yog tias qhov txhab cuam tshuam rau lub paj hlwb cortex, tuaj yeem ua rau qaug dab peg nyob rau theem mob cerebrovascular.Feem ntau, qhov tshwm sim siab tshaj plaws yog nyob rau hauv 1 hnub tom qab tus kab mob, thaum cov kab mob cerebrovascular nrog qaug dab peg raws li thawj qhov tshwm sim tsis tshua muaj.

Yuav kho Cerebral Infarction li cas?

Kev kho tus kab mob yuav tsum paub txog kev kho mob ntshav siab, tshwj xeeb tshaj yog nyob rau hauv cov neeg mob lacunar infarction hauv lawv cov keeb kwm kho mob.

(1) Lub Sijhawm Mob

a) Txhim kho cov ntshav ncig ntawm cheeb tsam cerebral ischemia thiab txhawb kev rov ua haujlwm ntawm cov hlab ntsha sai li sai tau.

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c) Tsawg molecular hnyav dextran tuaj yeem siv los txhim kho microcirculation thiab txo cov ntshav viscosity.

d) Diluted ntshav

f) Thrombolysis: streptokinase thiab urokinase.

g) Anticoagulation: siv Heparin lossis Dicoumarin los tiv thaiv thrombus dilation thiab thrombosis tshiab.

h) Dilation ntawm cov hlab ntsha: Nws feem ntau ntseeg tias cov nyhuv ntawm vasodilators tsis ruaj khov.Rau cov neeg mob hnyav uas muaj zog intracranial siab, qee zaum nws tuaj yeem ua rau tus mob hnyav dua, yog li ntawd, nws tsis pom zoo siv thaum ntxov.

(2) Lub sijhawm rov qab los

Txuas ntxiv mus ntxiv dag zog rau kev cob qhia kev ua haujlwm ntawm tes taw tes tuag taw thiab hais lus ua haujlwm.Cov tshuaj yuav tsum tau siv nrog rau kev kho lub cev thiab acupuncture.